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Pharmacological Management of Seizures & Cortical Localization, Drug…
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- Drug Mechanisms & Key Adverse Effects
- MOA: Presynaptic voltage-gated Na+ channel blocker
- Key Side Effect: Gingival hyperplasia
- MOA: Presynaptic voltage-gated Na+ channel blocker
- Key Side Effect: Stevens-Johnson Syndrome (especially with HLA-B*1502)
- MOA: Presynaptic voltage-gated Na+ channel blocker
- Key Side Effect: Severe life-threatening cutaneous rash (SJS/TEN)
- MOA: Multi-target (blocks Na+/Ca2+ channels and enhances GABA)
- Key Side Effect: Teratogenicity (neural tube defects / spina bifida)
- MOA: Binds SV2A synaptic vesicle protein to inhibit glutamate release
- Key Side Effect: Neuropsychiatric and behavioral disturbances
- MOA: Blocks thalamocortical T-type Ca2+ channels
- Key Side Effect: Gastric distress (nausea and abdominal pain)
- MOA: Increases GABA-A chloride channel opening frequency
- Key Side Effect: Respiratory depression and severe sedation
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