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Acute Kidney Injury (AKI) - Coggle Diagram
Acute Kidney Injury (AKI)
Etiology
severe deyhydratrion
Nephrotoxic drugs
NSAIDS
Amnioglycosides
Systemic Shock
Pathophisiology
Sudden decline in GFR (Glomerular Filteration Rate)
Renal Hypoperfusion
Toxic tubular injury
Signs
Sudden oliguria (<400 ml/day)
Quick rise in creatinine levels
Elevated BUN (blood urea nitrogen)
Hyperkalemia (high potassium)
Metabolic acidosis
Symptoms
Rapid onset peripheral edema
Confussion
Drowsiness
Shortness of breath caused by fliud overload
Nausea
Vomiting
Fatigue
Weakness
Diagnosis
KDIGO diagnostic criteria evaluaiton
Renal bladder ultrasound
Serum electrolyte monitoring
Urinalysis
Treatment
IV resuscitation
Discontinue nephrotoxic agents
Diuretic therepy
Medications to lower potassium
Prognosis
Possible complete recovery for kidneys
Risk of progression to CDK (Chronic Kidney Disease)
Critical illness patients have a higher risk for mortality
Increase chance for future AKI
Potential for permanent nephron loss