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WHITE LESIONS 3 - Coggle Diagram
WHITE LESIONS 3
Oral Lichen Planus
Etiology
Drugs
Dental materials
Infectious agents
Autoimmunity
Genetic
Immunodeficiency
Clinical Features
Females > males
Wickham striae
Middle-aged adults
Posterior buccal mucosa
Clinical Patterns
Papular
Plaque
Erosive
Bullous
Atrophic
Reticular
Diagnosis
Biopsy
Direct immunofluorescence
Clinical examination
Histopathology
Hyperorthokeratosis
Civatte bodies
Hyperparakeratosis
Band-like lymphocytic infiltrate
Management
No treatment if asymptomatic
Antifungal therapy if candidiasis present
Patient education
Oral Submucous Fibrosis
Etiology
Areca nut
Tobacco
Betel quid
Pathogenesis
Fibrosis induced by areca nut
Carcinogenesis mainly due to tobacco
Clinical Features
Trismus
Tongue stiffness
Burning sensation
Leukoplakia
Fibrous bands
Erythroplakia
Marble-like mucosa
Histopathologic Features
Epithelial atrophy
Diffuse fibrosis
Hyperkeratosis
Dysplasia
Treatment
Surgical excision of fibrous bands
Lifelong physiotherapy
Intralesional corticosteroids
1. Lupus Erythematosus
Types
Chronic cutaneous lupus erythematosus (CCLE)
Oral lesions usually associated with skin lesions
Ulcerated/atrophic erythematous central zone
Similar to erosive lichen planus
White fine radiated striae
Painful with acidic or salty foods
Subacute cutaneous lupus erythematosus (SCLE)
Photosensitivity
Skin lesions
Intermediate between SLE and CCLE
Systemic lupus erythematosus (SLE)
Arthritis
Weight loss
Fever
Fatigue
Butterfly rash
Multisystem disease
Oral lesions (5–25%)
Erythema
Hyperkeratosis
Pain
Xerostomia
Ulceration
Stomatodynia
Gingivae
Candidiasis
Buccal mucosa
Periodontal disease
Palate
Histopathologic Features
Basal cell degeneration
Subepithelial lymphocytic infiltration
Hyperkeratosis
PAS-positive basement membrane
Diagnosis
Direct immunofluorescence
IgM, IgG, C3 deposition
Treatment
Topical corticosteroids
Hairy Leukoplakia
Etiology
HIV infection
Immunosuppression
AIDS
EBV-related lesion
Clinical Features
Lateral border of tongue
Vertical white bands
White plaque
Shaggy surface
Histopathologic Features
Balloon cells
Nuclear beading
Acanthosis
No dysplasia
Parakeratosis
Treatment
Retinoids
Podophyllum resin
Antiviral drugs
Surgery
Usually no treatment
Cryotherapy
Actinic Cheilosis
Etiopathogenesis
Chronic UV light exposure
TP53 mutation
Risk Factors
Smoking
Immunosuppression
Elderly men
Outdoor workers
Clinical Features
Fissuring
Rough scaly lesion
Dryness
Leukoplakia
Atrophy
Chronic ulceration
Treatment
Biopsy
Vermilionectomy
Hyperplastic Candidiasis
Clinical Features
Raised
Irregular
Bilateral
Palpable
White patch
Diagnosis
Candidal hyphae
Resolution after antifungal therapy