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PHYSIOLOGY: SPRINT - Coggle Diagram
PHYSIOLOGY: SPRINT
Physiological Adaptations to RST
Phosphocreatine system efficiency
SPT causes significant depletion of PCr stores- contributing to onset of fatigue
GAITANOS (1993)
RST has shown enhanced PCr resynthesis through improvements in oxidative metabolism and oxygen capacity
enhanced PCr resynthesis delays onset of fatigue and has led to faster mean time of RPA
(BISHOPS ET AL., 2011)
Glycolytic buffering + metabolite clearance
as sprint duration increases or recovery time decreases, there is greater accumulation of lactate and H+ ions in the muscle. this can impair
glycolytic enzyme activity, calcium handling, excitation-contraction coupling, force production
RST improves muscles ability to tolerate and buffer the metabolic changes and maintain muscle pH which delays the onset of fatigue leading to better mean repeated sprint times
(Bogdanis 1996)
Allen et al., (2008)
enhanced H+ / lacatate clearance during rest - improved bloodflow and capillarisation
Performance adaptations to RST
increased sprint speed
increased peak power
improved recovery time between rest periods
WHY?
greater force production
motor unit recruitment
ATP resynthesis
(ross et al., 2001)
reduced perception of fatigue