Referencias:
-Brack, M., et al. (2020). Autonomic dysfunctions after spinal cord injury. Spinal Cord, 58(12), 1184–1193.
-Fehlings, M. G., & Sekhon, L. H. (2001). Epidemiology, demographics, and pathophysiology of acute spinal cord injury. Spinal Cord, 39(7), 355–365.
-Fleming, J. C., et al. (2006). The cellular inflammatory response in human spinal cords after injury. Brain, 129(12), 3241–3259.
-Lipton, S. A., & Rosenberg, P. A. (1994). Excitatory amino acids as a final common pathway for neurologic disorders. The New England Journal of Medicine, 330(9), 613–622.
-Rowland, J. W., et al. (2008). Current status of acute spinal cord injury physiophysiology and emerging therapies: promise on the horizon. Neurosurgical Focus, 25(5), E2.
-Tator, C. H., & Koyanagi, I. (1997). Vascular mechanisms in the pathophysiology of human spinal cord injury. Journal of Neurosurgery, 86(3), 483–494.
-Xiong, Y., et al. (2007). Role of peroxynitrite in secondary oxidative damage after spinal cord injury. Journal of Neurotrauma, 24(12), 1845–1861.