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Acute Respiratory Distress Syndrome, Early Exudative Stage (Stiegel, 2024)…

- - - - 43% mortality rate (Diamond et al., 2024)
- - - - Pathogen invades host (Kumar et al., 2021)
        
        Host immune response (Kumar et al., 2021)
        
        Proinflammatory mediator release (TNF & Interleukins) (Kumar et al., 2021)
        
        Tissue Damage
  - - - Duct Obstruction, Defective Intracellular Transport (Kumar et al., 2021)
        
        Acinar Cell Injury (Kumar et al., 2021)
        
        Interstitial Inflammation & Edema, Proteolysis, Fat Necrosis, Damage to vessel walls (Kumar et al., 2021)
    - - Antibodies and lipids stored in blood products activate neutrophils (Cho et al., 2023)
        
        Capillary leakage of intracellular content-releasing proteases and elastase (Cho et al., 2023)
        
        Oxidation (Cho et al., 2023)
        
        Pulmonary Edema (Cho et al., 2023)
- - - - Secretion of mediators (TNF) (Kumar et al.,2021)
  - - - Endothelial Activation (Kumar et al.,2021)
        
        Endothelial cells express increased levels of adhesion molecules, procoagulant proteins, and chemokines (Kumar et al.,2021)
        
        Adhesion and extravasation of neutrophils (Kumar et al.,2021)
        
        Neutrophils adhere to activated endothelium and move to alveoli (Kumar et al.,2021)
        
        Release of inflammatory mediators (proteases, ROS, cytokines)(Kumar et al.,2021)
        
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- - - - Respiratory Distress (Siegel, 2024)
  - - - Altered Mental Status (Siegel, 2024)
- - - - Widespread patchy opacities in dependent lung zones often referred to as "ground glass" (Siegel, 2024)
        
        Becomes more consolidative (Siegel, 2024)
    - - Bilateral diffuse alveolar opacities with dependent atelectasis (Siegel, 2024)
- - - - Lasts for 2-3 weeks (Stiegel, 2024)