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Non cancer ovarian pathologies - Coggle Diagram
Non cancer ovarian pathologies
Follicle/Luteal cysts
very common
physiologic variants
origin
unruptured graafian follicles
ruptured and sealed follicles
often multiple
develop immediately below serosal lining
usually small
lined by granulosa/luteal cells when small
as fluid accumulates pressure may cause atrophy of these cells
may rupture
intraperitoneal bleeding
pain
Polycystic ovaries
Stein Leventhal syndrome
multiple cystic follicles
produce excess estrogens and androgens
presentation
oligomenorrhoea
hirsutism
infertility
obesity
ovaries
twice normal size
grey white
smooth outer cortex
multiple subcortical cysts
biochemical abnormality
excess androgens
proposed that converted to estrone
in peripheral fat depots
inh. FSH secretion by pituitary
^ through HP axis
high conc. of LH
low conc. of FSH
reduced follicles
infertility
ovarian torsion
infrequent
significant cause of acute lower abdominal pain in women
classically in enlarged ovary
60% on right side
risk factors
children
developmental abnormalities
pregnancy
enlarged corpus luteum
ovulation induction
ovarian tumours
50-60% of cases
presentation
70-75% <30yrs
20% in pregnancy
17% pre-menarchal/post-menopausal
sudden onset
severe unilateral lower abd. pain
worsens intermittently
early dx +treatment
excellent prognosis
delayed dx
infarction
necrosis
ovarian salvage rate
10%