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Valvular diseases - Coggle Diagram
Valvular diseases
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Aortic regurgitation
AR causes LV volume overload, CO maintained by increasing SV at expense of LV end-diastolic volume
LV dilation - myocyte function decrease - LV failure --> CHF
Acutely- immediate surgical intervention
Symptoms
Dyspnea, orthopnea, palpitations, angina/chest pain, possible symptoms of underlying cause
Diagnosis
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Auscultation
Low intensity and high pitched decrescendo type murmur, over left sternal border or right second IC space, early diastile
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Physical examination
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Corrigan's sign- visible carotid pulsation, 'dancing neck arteries'
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Causes
Annulus dilation, aneurysm and dissection
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Valvular
chronic
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Rheumatic - fibrosis, fusion, retraction of cusps
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Prognosis
chronic type tolerated for many years, 5 yr survival rate around 75%, 10 yr around 50%
worsens as symptoms dvlp
acute severe AR is associated with high mortality from LV failure, early surgical intervention indicated
Mitral stenosis
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Symptoms
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Dyspnea on exertion, orthopnea, PND
Hemoptysis, hoarseness, RV failure symptoms and dysphagia
LA dilation and hypertrophy, and RV hypertrophy/dilation
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at chordae, cusps, commissures
increased resistance of flow from LA-LV, elevated LA pressure, causes backward pulmonary venous HTN - secondary pulmonary arterial HTN - RV hypertrophy and failure
Diagnosis
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Auscultation
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Graham steel murmur (due to pulmonary HTN)
early blowing diastolic murmur, heard along left border of sternum due to functional regurgitation through pulmonic valve
Mid-diastolic crescendo murmur (low-pitch, rumbling) with opening snap
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Treatment
Asymptomatic
Sinus rhythm, BB, Ca-channel blockers
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Symptomatic
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AF: BB, Ca-channel blockers or digoxin
Anticoagulants
Severe: indication for surgery (ACC/AHA guidelines)
Percutaneous mitral balloon valvotomy (PMBV)
Closed valvotomy - separation of fused cusps
Open valvotomy w/ cardiopulmonary bypass (pref)
Mitral valve replacement
Aortic stenosis
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Symptoms
Exertional dyspnea / CHF, angina, syncope
Diagnosis
Auscultation
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Mid-systolic crescendo-decrescendo murmur- aortic area in full expiration, extends to carotids
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Physical examination
Pulsus parvus et tardus - small volume, slow rising pulse
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TTE
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Confirms dg- calcified valve with restricting opening, or showing congenital abnormalities and aortic pathology
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Treatment
Surgery- aortic valve replacement if symptomatic, positive stress test and EF <50%
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Poor prognosis without surgery
If surgery not done:
angina - 50% die within 5 years
syncope - 50% die within 3 years
congestive HF - 50% die within 2 years
Mitral regurgitation
Symptoms
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Chronic MR: fatigue, exertional dyspnea, orthopnea, systemic embolization.
Palpitations: premature atrial beats or AF
Later stages: pulmonary HTN/ RV failure
Causes
Chronic - gradual enlargement of LA, LV enlargement and hypertrophy, AF (common), thromboembolism and risk for IE
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Acute - no compensation of LA and LV, immediate pulmonary edema and cardiogenic shock (biventricular failure)
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Diagnosis
ECG
LA enlargement, LV hypertrophy and RA enlargement in pulmonary HTN
AF (common)
CXR
Cardiomegaly, calcified mitral valve, pulm, venous congestion
Auscultation
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Pansystolic plateau murmur, at apex in lateral decubitus
TTE
confirms dg (+ catheterization)
hyperdynamic LV, possible vegetations in IE
color doppler to detect and quantify the MR, assess LV size and function (EF, End-systolic diameter, End-diastolic diameter)
CW Doppler: assess velocity of regurgitant jet and estimate PA pressure
Prognosis
Poor outcome in symptomatic pt, 33% survival at 8 years without surgical intervention
Death usually due to heart failure or arrhythmia related
Treatment
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Invasive
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Ind for acute type, and symptomatic pt and asymptomatic with severe MR (LV enlargement or LV systolic dysfunction)
Tricuspid valve stenosis
Symptoms: fatigue, anorexia, peripheral edema
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