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Acute Pancreatitis - Coggle Diagram
Acute Pancreatitis
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Local: Pancreatic necrosis, abscess, pseudocyst formation
Renal: Acute kidney failure, electrolyte imbalances
Pulmonary: ARDS, pleural effusions, atelectasis
Cardiovascular: Hypotensive, septic, or hemorrhagic shock
Metabolic: Hyperglycemia, metabolic acidosis, hypocalcemia
Gastrointestinal: GI bleeding, ileus, splenic infarction.
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Lifestyle modifications: Low-fat diet, alcohol cessation, and management of underlying metabolic disorders.
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Low-fat, high-protein diet introduced gradually when pain resolves
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Metabolic disorders (hypertriglyceridemia, hypercalcemia, renal failure)
Infections (mumps, viral hepatitis, Mycoplasma, CMV, Candida, TB) :
Medications (azathioprine, sulfonamides, tetracyclines, valproic acid).
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Hypocalcemia, leukocytosis, hyperglycemia
Elevated bilirubin, AST, and alkaline phosphatase in biliary involvement
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Ultrasound, CT scan, or MRI to detect pancreatic inflammation, gallstones, or necrosis.
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Fever, hypotension, and jaundice
Grey Turner’s sign (flank bruising) and Cullen’s sign (periumbilical ecchymosis) indicating hemorrhagic pancreatitis.
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Acute pancreatitis is an inflammatory condition of the pancreas caused by premature activation of digestive enzymes, leading to auto-digestion. It is classified into:
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Severe (Necrotizing): Associated with infection, tissue necrosis, hemodynamic instability, and multi-organ dysfunction.
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Digestive enzymes (e.g., trypsin, elastase, lipase) activate prematurely, leading to auto-digestion.
Enzymes trigger inflammation, vascular permeability increase, hemorrhage, and necrosis.
Systemic complications arise from enzyme release into circulation, leading to shock, multi-organ failure, and systemic inflammatory response syndrome (SIRS).
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On admission: Age >55, hypotension, abnormal lung findings, abdominal mass
Within 48 hours: Hematocrit drop >10%, leukocytosis, hyperglycemia, hypocalcemia, azotemia
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