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PEMFT - Coggle Diagram
PEMFT
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Mechanisms of action
-An electric current in the applicator which drives an electromagnetic field.
-This induces small electric field changes within the target tissue, responsible for physiological and therapeutic effects.
Globally- optimises the bodies ability to repair itself, re-establishes normal currents & cellular voltage.
- Ca2+ binds to calmodulin = bursts of nitric oxide (NO). - NO upregulates anti-inflammatory responses, vasodilation + growth factors for neovascularization, tissue regeneration / remodelling.
Specifically...
Vasoconstriction (first 72hrs)
- Injured cell - disrupted ion balance = decreased function & death.
- electromagnetic field moves electrons in/out = restores balance.
- PEMF kick starts adenosine receptor expression = reduces inflam mediators - reduced pain and inflammation.
- constriction of capillaries also reduces oedema formation.
Vasodilation
- Mast cells stimulated to produce Histamine release
- Detected by hypothalamus & releases Bradykinins
- Bradykinins relax smooth muscle of vessel walls.
- Increases circulation = increase of nutrient rich blood & O2 for cell function & removal of waste products.
- NO also causes local vasodilation .
Pain relief
- Disrupts the brains ability to transmit pain by decreasing the nerve cell potential from -70 (normal) to a hypo-polarisation level of -90mV.
- Cell voltage too low when the pain signal is detected, for the synapse to trigger the release of the chemical transmitters
- May also stimulate release of endogenous opioids.
- Inflammation- pressing on nocioceptors = causing pain. PEMF cx release of NO (anti inflammatory mediator).
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Neuro
- Vasodilation & pain relief as across.
- Upregulates neurotransmitters, promoting nerve signalling & re establishing cellular currents.
Target tissue
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Anecdotally:
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MSK injury, soft tissue injury and repair
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