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`PATHOLOGY—CELLULAR INJURY - Coggle Diagram
`PATHOLOGY—CELLULAR INJURY
Cellular adaptations
Hypertrophy
Hyperplasia
Atrophy
Metaplasia
Dysplasia
Cell injury
Reversible cell injury
Irreversible cell injury
Apoptosis
intrinsic (mitochondral pathway)
Extrinsic (Death receptor) pathway
Necrosis
Coagulative
Liquefactive
Caseous
Fat
Fibrinoid
Gangrenous
Inflammation
Cardinal signs
rubor redness
Tumor (swelling)
Dolor (pain)
Functio laesa (loss of
function)
Systemic manifestations (acute-phase reaction
Fever
Leukocytosis
increase plasma acute-phase
proteins
Acute phase reactants
More FFiSH in the C (sea).
Granulomatous
inflammation
HISTOLOGY
MECHANISM
ETIOLOGIES
Chronic inflammation
STIMULI
MEDIATORS
OUTCOMES
Ischemia
Types of infarcts
Red infarct
Pale infarct
Free radical injury
Types of calcification
Dystrophic calcification
Metastatic calcification
Lipofuscin
Amyloidosis systemic
Primary amyloidosis
Secondary
amyloidosis
Dialysis-related
amyloidosis
Amyloidosis Localized
Alzheimer disease
Type 2 diabetes
mellitus
Medullary thyroid
cancer
Isolated atrial
amyloidosis
Systemic senile (age related)
amyloidosis
Hereditary
Familial amyloid
cardiomyopathy
Familial amyloid
polyneuropathies
Erythrocyte
sedimentation rate
Acute inflammation
STIMULI
MEDIATORS
COMPONEN TS
OUTCOMES
Leukocyte
extravasation
Wound healing
Tissue mediators
PHASE OF WOUN D HEALING