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Convulsive conditions :seizures - Coggle Diagram
Convulsive conditions :seizures
pathophysiology
An imbalance of excess excitation and decreased inhibition initiates the abnormal electrical activity. These electrical paroxysmal depolarization shifts ( PDS)
Seem to trigger epilepti form activity. Increased activation or decreased inhibition of such discharges could result in seizures. The part of the brain affected often reflects in the clinical signs or symptoms of the seizures.
Neurotransmitter are undoubtedly involved. Glutamate is the most common excitatory neurotransmitter, and gamma- aminobutyric acid (GABA) is an important inhibitory neurotransmitter
On cellular level seizures start with the excitation of susceptible cerebral neurons, which leads to synchronous discharge of progressively larger groups of connected neurons.
risk factors
hyperventilation
physical stress
fatigue
hypoglycemia
elderly
genetics
diagnoses and treatment
surgery
dietary changes
control seizures
anti epileptic drugs AED
physical exam
clinical manifestation
headache
confusion
memory loss
actual seizures
definition
is a brain disorder characterized by an enduring predisposition to generate epileptic seizure
causes
genetic syndrome
duplication syndromes
metabolic disorders
mitochondrial diseases
single gene mutations