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Psychopathology - Coggle Diagram
Psychopathology
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phobias
characteristics
behavioural : Panic- screaming or running away, avoidance - conscious effort to avoid.
emotional: anxiety- unpleasant state of high arousal, disproportionate to the threat.
fear- short lasting , more intense.
emotional response is unreasonable / disproportionate to the threat.
cognitive: selective attention to the phobic stimulus- cant look away. irrational beliefs- unfounded beliefs.
cognitive distortions - unrealistic
behavioural explanation
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acquisition by classical conditioning: UCS links to NS, the both produce UCR (fear) , now called the CR. like little albert played with rats (NS) , heard a loud noise (UCS) then the rat (now CS) produces gear response.
maintenance by operant conditioning: avoidance of phobic stimulus negatively reinforced by anxiety reduction, so the phobia is maintained.
evaluation
real world application; phobias successfully treated by preventing avoidance, as suggested by the model.
cognitive aspects of phobias: fails to account for cognitive aspects of phobias, e.g. irrational fears.
phobias and traumatic experiences: 73% of people with a dental phobia had past trauma, and in a control group, only 21% had past trauma. however, not all cases of phobias follow bad experiences and vice versa.
learning and evolution: two- prosses model explains individual phobias, but evolutionary approach explains general aspects of phobias.
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flooding
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evaluation
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traumatic: rated as more stressful than systematic sensitisation , lack of informed consent and higher attrition rates.
symptom substitution: occurs if the cause of the phobia is not tackled, someone with a phobia looses one through flooding but gains or inflated another existing phobia
depression
characteristics
behavioural : activity levels - lethargy or agitation \, disruption to eating and sleeping behaviour , aggression and self harm and irritability.
emotional : lowered mood. anger towards self and others leading to behavioural change, lowered self esteem , self loathing.
cognitive: poor concentration - difficulty making decisions. attending to and dwelling on the negative- half- empty glass mentality. absolutist thinking.
Becks theory
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negative self schema: negative information about ourselves is assessed whenever we encounter a self -relevant situation .
the negative triad: negative views of the self, world and future.
evaluation
research support: research shows cognitive vulnerability proceeds depression (Clark and Beck , Cohen et al in teens)
real world application : identify cognitive vulnerability to screen those at risk of depression, then target variabilities with CBT.
a partial explanation: explains patterns of cognition, but cannot easilty explain extremes of anger or hallucinations and dilutions
Ellises ABC model
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B = beliefs: beliefs lead us to over react to the activating event, e.g. that life must always be fair (utopianism) or we must succeed (musturbation)
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evaluation
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reactive and endogenous depression ; only explains reactive depression, does not explain cases that do not have activating events.
ethical issues: ABC moel places the balance on the depressed perosn (victim blaming ) but therapy derived from the model (REBT) does create resilience.
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OCD
characteristics
behavioral: compulsions are repetitive. compulsions are performed to reduce anxiety. avoids situations that trigger anxiety.
emotional : anxiety and distress created by compulsions or obsessions . following depression depression, guilt and disgust- directed at something such as dirt or oneself.
cognitive: obsessive thoughts, e.g. about germs, cognitive coping strategies like meditating, insight into excessive anxiety- may include catastrophic throughs and hypervigilance.
genetic
candidate genes: genes that may be involved in producing symptoms of OCD , e.g. 5Ht1-d beta
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evaluation
research support: 68% MZ twins and 31% of DZ have OCD , OCD 4 times more likely if you have a family member with it .
environmental risk factors: over half OCD clients in one sample experienced a traumatic event , and OCD was more severe.
animal studies: candidate genes have been found in mice , but can be generalise between humans and animals.
neural explanation
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evaluation
research support antidepressants that work on the serotonin system elevate OCD, biological conditions (prisons or example) have similar symptoms to OCD.
no unique neural system: the apparent serotonin- OCD link may just be co -morbidity with depression- the depression directs serotonin.
correlation and causality: dysfunction of neural systems may cause ocd but most evidence is correlational , so could be vice versa.
drug therapy
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combining SSRi's with other treatments : SSRI's CBT offers best effectiveness, plus maybe other drugs.
alternatives to SSRI's : tricyclics , act on serotonin plus other systems, or SNRI's.
evaluation
evidence of effectiveness: 17 studies are showed SSRI's more effective than placebos. however psychological therapies alone are likely to be more effective than drugs for OCD.
cost effective and non disruptive: relatively cheap for NHS and don't involve time spent going to therapy sessions.
serious side - effects : SSRIs may lead to indigestion, blurred vision and loss of sex drive, worse for clomipramine
biased evidence: drug researchers sponsored by drug companies , biased results, but still best available evidence and psychological therapies research may be biased too