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Neuro/cognition: ischemic CVA - Coggle Diagram
Neuro/cognition: ischemic CVA
Risks
smoking
DM
HTN
genetics
A-fib
excessive alcohol/cocaine use
obesity
S&S
one sided facial droop
slurred speech
One sided weakness
sudden ataxia
one sided vision impairment
complications
dysphagia, aphasia, dysarthria
hemiparesis
hemineglect
hemiplegia
additional strokes
cognitive impairments
Treatment
Stents/clot disruption if clot is in accessible area
prevent further strokes, usually with blood thinners/ lifestyle change depending on risk factors
Rapid treatment can limit the area of infarction, treatment focuses on maintaining ABCs and tPA once hemorrhagic stroke is ruled out and no contraindications are detected.
Recovery from stroke promotes avoiding further complications and rehabilitation
diagnostics
Labs and tests to find underlying mechanism of stroke, CBC, clotting factors, lipid panels etc....
CT scan to rule out hemorrhagic stroke, then MRI (preferred method) or angiography to determine area of ischemia/infarction
Cerebral vascular flow is blocked or disrupted by a precipitating event in a cerebral or carotid artery. As a result neurons do not receive adequate O2 and become ischemic. As this occurs cellular metabolism fails causing the failure of the Na/K pump leading to cellular edema in the area of ischemia. Infarction then occurs as cells go prolonging time without receiving fresh blood. a penumbra develops around this infraction zone and can cells located here can be saved if perfusion is restored. The symptoms seen in CVA are dependent on the location of the blockage, with more symptoms presents with more proximally located thrombi/embolisms.