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Neuro/Cognition: HEM CVA - Coggle Diagram
Neuro/Cognition: HEM CVA
Pathophysiology/Etiology
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Dilation or weakness in cerebral vessels increases vulnerability. Blood vessel ruptures from pressure build-up and leaks blood into surrounding brain tissue called subarachnoid hemorrhage.
Accumulation of fluid in the brain cavity increases intercranial pressure. Exposure to blood impairs cerebral metabolism. Compression of small blood vessels causes ischemia.
An open leak in the closed vascular system decreases perfusion pressure and impairs blood flow through the brain. Vasoconstriction occurs in attempt to increase pressure, causing vasospasm and further tissue injury.
Nerve cells switch to anaerobic metabolism due to oxygen deprivation and blood pooling. Lactic acid build-up and ion imbalance causes further tissue injury. Nerve cells stop functioning and cause neurological effects.
Loss of blood into cerebral space decreases vascular volume and causes hypovolemia. Heart rate increases to maintain cardiac output. Systemic hypoperfusion of all organs causes low blood pressure.
Symptoms
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Numbness or weakness on face, arm, or leg
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Collaborative Treatment
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Stop anticoagulants, correct INR
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