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4 Language impairments in ASD, One is that this may lack external…
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- One is that this may lack external validity/not be generalisable, because this study of FOXP2 gene was based on 1 family in London, and this was a very small sample, and is unlikely to be generalisable. Studies show that many other people with language impairments just like the KE family have NO mutation of the FOXP2 gene, thus it's unlikely that the findings of the KE family can be generalisable
- There may be grandiose claims about this FOXP2 gene, as the FOXP2 gene mutation can affect several other brain/body functions, and thus may not be a "speech gene" as it is not solely implicated in speech, thus other humans can have a mutation of the FOXP2 gene but have NO language impairment. Studies show that mices who had a mutation of the FOXP2 gene had lots of changes to their lungs and brain. (but tbh, how are they meant to assess the language of mice - maybe you can just say other humans with this mutation had lung/brain changes).
- there's also this one bit, where in the Atlantic article, you can say that the support for the FOXP2 gene generally has come from mouse studies, where they show that mutations of this gene in mouse studies have led to changes in pitch of mouse vocalisation, but then you can criticise this by saying acc there are many diff confounds that could've led to this bc the FOXP2 gene may have influenced many other organ systems which could've led to a change in vocalisation - but tbh right, it's still sort of supporting the idea that FOXP2 is implicated in language, it also goes against your use of evidence about the mice who had changes in lungs and brain (bc its showing that mice studies shouldn't be trusted as they're not internally valid), and also, the question might specifically be about the KE family & NOT about supporting evidence...