Please enable JavaScript.
Coggle requires JavaScript to display documents.
Myocardial infarction - Coggle Diagram
Myocardial infarction
-
Pathophysiology
It is caused by prolonged aschaemia of the myocardium when a myocardial cell necrosis is not reversible.
It happened after the myocardial oxygen supply and demand is not balanced, which is most often caused by plaque rupture with thrombus formation in a coronary vessel, resulting in an acute reduction of blood supply to a portion of the myocardiam
-
Diagnostic studies
-
-
-
-
-
Decreased cardiac output related to altered conduction of impulses secondary to necrosis of the conducting system evidenced by changes in rate and rhythm of the heart and blood pressure.
Anxiety related to perceived threat of death evidences by expression of fear and uncertainly about the future.
-
Complications
-
Haemorrhage, systemic or intracranial, which tis treated with tranexamic acid.
-
-
Cardiac rupture but it is a complication of late thrombosis which is pre treated with IV beta blocks .
-
-
-
-
-
-
-
-
Nursing intervention
-
-
-
-
Administration
-
Aspirin, specifically disprin may be prescribed to reduced the cougulability of the blood
Nitrate as prescribed to dilate the coronary blood vessels and increase blood flow to the myocardium
B-adrenergic blocking agents such as Atenolol and Propranolol are prescribed to reduce excitability and contractility of myocardium
-
Calcium channel blockers to relieve the coronary artery spasm and increase blood flow to the myocardium because coronary artery spasm can increase ischaemia following myocardial infarction
-
An opioid analgesic, specifically morphine- besides being pain killers but morphine has a vasodilator and calming effect, which is beneficial.
-
-
-
-