Innate immunity II

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Innate immune response

Inflammation and cell recruitment

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Stimulation of adaptive immunity

Phagocytosis

Phagocytosis represents an early and crucial event in triggering host
defenses against invading pathogens.

Phagocytosis comprises a series of events

microbicidal function of immune cells.

antigen
presentation, allowing the induction of acquired immunity.

defense mechanism

inflammatory mediators and inflammatory cells recruitment.

Acute innate cytokines

(vasoconstriction →vasodilation)

redness, increased heat, swelling, pain, loss of function

Increase in vascular permeability

Extravasation of leukocytes

Phagocytosis

Outcome of inflammatory response

Leukocyte recruitment cascade

:Interactions of various cytokines

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IL-6, interleukin-6

Acute phase proteins:

LBP, LPS-binding protein

CRP, C-reactive protein

SAA, serum amyloid

Cytokines and their functions

:Pro-inflammatory vs Anti-inflammatory cytokines

Acute phase proteins (APPs)

specific response

systemic reaction

These proteins are secreted mainly by hepatocytes.

IL-6, TNF, and IFN-γ, are
produced by macrophages, monocytes, and other cells.

IL-6 is the
major inducer of APP.

APPs: C-reactive protein (CRP)

C-reactive protein (CRP) is a non-specific APP that binds to phosphocholine
on microorganisms.

enhancing phagocytosis
of macrophages and helping in complement binding of damaged cells.

increased rapidly within 2 hours of acute insult, rise above
normal limits within 6 hours, and peak at 48 hours.

Chronic inflammation

Granulomatous inflammation

Anti-viral immunity

pDCs detect RNA and DNA viruses with two endosomal sensors, TLR7 and TLR9,

Cellular sources of IFN-I vary with the type of viral infection.

pDCs are bone marrow-derived cells that secrete large amounts of IFN-I
(primary source of IFN-I for antiviral responses).

Cellular sources of IFN-I during virus infections

Virus

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Molecular sensors involved in virus recognition

IFN-I signaling pathway.

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Effects of IFN-I on cells during virus infections

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T-cell stimulation and T helper 1 (TH1)/TH2-cell
polarization require three dendritic cell-derived signals

Th1 response

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Th2 response

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Professional antigen presenting cells (APCs)

Summary