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MULTIPLICATION - Coggle Diagram
MULTIPLICATION
BACTERIOPHAGE
VIRAL REPLICATION
SYNTHESIS
ENTRY OF THE NUCLEIC ACID CAUSES THE CESSATION OF HOST CELL DNA REPLICATION AND PROTEIN SYNTHESIS. THE HOST CELL IS USED AS THE MACHINERY FOR VIRAL REPLICATION AND SYNTHESIS OF VIRAL PROTEIN
MATURATION
AS THE HOST CELL PRODUCES NEW PHAGE PARTS, THE PARTS SPONTANEOUSLY ASSEMBLE INTO BACTERIOPAGE
PENETRATION
PENETRATION IS AN ENTRY OF VIRIONS OR THEIR GENOME INTO THE HOST CELL. BACTERIOPHAGE HAVE A MECHANISM FOR INJECTING THEIR NUCLEIC ACID INTO THE HOST WHICH ELIMINATES THE NEED FOR UNCOATING
ADSORPTION
ADSORPTION INVOLVE ATTACHMENT OF SPECIFIC TAIL FIBERS OF BACTERIA'S CELL WALL, BACTERIOPHAGE HAVE SPECIALIZED STRUCTURE FOR ATTACHING TO BACTERIAL CELL WALLS
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CLASSIFICATION OF VIRUS
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TEMPERATE PHAGE
THE PHAGE THAT ARE ABLE TO UNDERGO BOTH LYTIC AND LYSOGENIC CYCLE, THE STATE WHERE THE DNA INTEGRATE INTO THE HOST DNA.
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ANIMAL VIRUS
VIRAL REPLICATION
SYNTHESIS
SYNTHESIS IS WHERE NEW NUCLEIC ACIDS, CAPSID PROTEINS AND OTHER VIRAL COMPONENTS ARE SYNTHESIZED
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MATURATION
ONCE ALL THE VIRAL NUCLEIC ACID, ENZYMES AND OTHER PROTEINS ARE COMPLETELY SYNTHESIZED, THE ASSEMBLY OF COMPONENTS INTO A COMPLETE VIRIONS IN BEGIN
PENETRATION OR UNCOATING
PENETRATION IS THE ENTRY OF VIRIONS OR THEIR GENOME INTO HOST CELLS MEANWHILE UNCOATING IS THE SEPARATION OF NUCLEIC ACID FROM PROTEIN COAT. THE NUCLEIC ACID IS RELEASED AS THE ENVELOP AND CAPSID DISSOLVED
TYPE
ENDOCYTOSIS
THE HOST CELL ENGULF THE ENTIRE VIRUS, ENCLOSED IT IN A VESICLES OR VACUOLE
DIRECT FUSION
THE VIRAL ENVELOPE DIRECTLY FUSED OR MERGED WITH THE CELL MEMBRANE AS IT RELEASE THE NUCLEOCAPSID INTO THE INTERIOR CELL
RELEASE
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VIRUS RELEASED
ENVELOPED VIRUS RELEASED THROUGH BUDDING OR EXOCYTOSIS FROM THE MEMBRANES OF CYTOPLASM, NUCLEUS OR ENDOPLASMIC RETICULUM
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ADSORPTION
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ADSORPTION IS THE ATTACHMENT OF THE VIRUS TO THE HOST CELL. THE INVASION BEGIN WHEN THE VIRUS ENCOUNTER A SUSCEPTIBLE HOST CELL, THE VIRUS THEN ADSORBS SPECIFICALLY TO THE MEMBRANE RECEPTOR BY MAKING AN EXACT FIT WITH THE SPECIFIC HOST MOLECULE. THE MEMBRANE RECEPTORS ARE USUALLY GLYCOPROTEIN, WHICH CAN BE RECOGNIZED BY THE SPIKE OR VIRAL RECEPTORS. THE HOST CELLS THAT LACK COMPATIBLE VIRUS RECEPTORS ARE RESISTANT TO THE VIRAL ADSORPTION AND INVASION
INFECTION
LATENT INFECTION
LATENT INFECTION IS AN INFECTION OF A CELL WHERE THE REPLICATION CYCLE IS NOT COMPLETED, BUT THE VIRUS GENOME IS MAINTAINED IN THE HOST CELL WITHOUT REPLICATING OR CAUSING HARM
COMPARISON
ANIMAL VIRUS
ANIMAL VIRUS ATTACH THE SPIKES, CAPSID OR ENVELOPE TO PLASM MEMBRANE PROTEIN DURING ADSORPTION, THE ANIMAL VIRUS UNDERGO EITHER ENDOCYTOSIS OR FUSION PROCESS DURING PENETRATION, IT ALSO UNDERGO ENZYMATIC REACTION OF VIRAL PROTEIN DURING UNCOATING THE ENVELOPE, ANIMAL VIRUS. THE SYNTHESIS OF VIRIONS HAPPEN IN CYTOPLASM (RNA VIRUS)AND NUCLEUS FOR (DNA VIRUS)
BACTERIOPHAGE
BACTERIOPHAGE ATTACH THE TAIL FIBERS TO THE CELL WALL PROTEIN DURING ADSORPTION, THE BACTERIOPHAGE INJECT THE VIRAL NUCLEIC ACID THROUGH THE BACTERIAL CELL WALL DURING PENETRATION, BACTERIOPHAGE HAS A SPECIALIZED MECHANISM TO INSERT THE NUCLEIC ACID INTO THE HOST CELL WHICH ELIMINATES THE UNCOATING PROCESS. SYNTHESIS OF VIRIONS HAPPEN IN CYTOPLSM
