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Uveitis (Clinical manifestations (Redness, Pain, Light sensitivity,…
Uveitis
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Pathophysiology
The mechanism for trauma is believed to be a combination of microbial contamination and accumulation of necrotic product at the site of injury thereby stimulating proinflammatory processes.
For infectious etiologies of uveitis, it is postulated that the immune reaction directed against foreign molecules or antigen may injure the uveal tract vessels and cells.
However genetic, traumatic, or infectious mechanisms are known to promote or trigger uveitis.
When uveitis is found in association with autoimmune disorders, the mechanism may be a hypersensitivity reaction involving immune complex deposition within the viral tract.
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Causes
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An infection such as cat stratch disease, herpes zoster, syphilis, toxoplasmosis, tuberculosis, Lyme disease.
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Treatment
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Systemic or oral administration of steroids, other immunosuppressant or anti metabolite drugs.
Nursing management
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Post-operative
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Instruct the patient to call a physician immediately if vision changes; continuous flashing light appear, redness, swelling or pain increase, type and amount of drainage increase or significant pain is not relieved by acetaminophen.
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