Migraine

Definition

Recurrent throbbing headache often preceded by an aura and associated with nausea, vomiting and visual changes

A migraine aura may affect the patients eyesight with visual phenomena such as fortification spectra, shimmering or scotomas, but may also result in pins and needles, dysphasia and rarely weakness of limbs and motor function

Epidemiology

In 90% onset is before 40 years

If onset > 50 then pathology should be sought

More common in FEMALES than males

Usually the severity of migraine decreases with advancing age

Most common cause of EPISODIC HEADACHE (recurrent)

Aetiology

Brain chemical imbalance may be cause

May be caused by changes in the brainstem and its interactions with the trigeminal nerve

No known specific cause but there are partial triggers (CHOCOLATE)

Cheese

Oral contraceptives

Orgasms

Lie-ins

Hangovers

Alcohol

Chocolate

Tumult

Exercise

Risk Factors

Female

Age - can occur at any age but majority have first migraine in adolescence

Strong genetic component thus family history

Pathophysiology

Cortical spreading depression - self-propagating wave of neuronal and glial depolarisation that spreads across the cerebral cortex is thought to cause the aura

Neurogenic inflammation leads to vasodilation and plasma protein extravasation - leading to pain that propagates all over cerebral cortex

Changes in brainstem blood flow lead to an unstable trigeminal nerve nucleus and nuclei in the basal thalamus

Clinical Presentation

Migraine without aura

Migraine with aura

There may be a prodrome that precedes the headache by hours/days - yawning, cravings and mood/sleep changes

General migraine features

Two of - unilateral, pulsing, moderate/severe pain in head, aggravated by routine physical activity

During headache at least one of - nausea and/or vomiting during headache, photophobia and phonophobia

Attacks last 4-72 hours

Not attributable to another disorder

At least 2 attacks

Aura precedes the attack by minutes and may persist during it

Unilateral, pulsatile headache

Visual - chaotic cascading, jumbling, distorting lines, dots or zigzags , scotoma, hemianopia

Somatosensory - paraesthesiae from fingers to face

At least 2 of: unilateral pain, throbbing-type pain, moderate to severe intensity, motion sensitivity

At least 1 of: nausea/vomiting, photophobia/phonophobia, normal examination with no other attributable cause

Differential Diagnosis

The visual and hemisensory symptoms must be distinguished from thromboembolic TIAs

Brain tumour and temporal arteritis

Sudden migraine may resemble meningitis or subarachnoid haemorrhage

Tension headache, cluster headache, medication over-use headache

Treatment

Diagnosis

Always examine - eyes (for papilloedema and other eye issues using fundoscopy), BP, head & neck (scalp, neck muscles and temporal arteries)

Exclude other causes

Mainly clinical diagnosis

Lab tests - CRP & ESR

Indications for neuroimaging (CT/MRI) - worst/severe headache (SAH), change in pattern of migraine, abnormal neurological exam, onset > 50 years, epilepsy, posteriorly located headache

Lumbar puncture indications - SAH (bad headache), severe rapid onset headache, progressive headaches, unresponsive headaches, NEUROIMAGING SHOULD PRECEDE

Acute - Don't offer ergots or opioids, TRIPTANS, NSAIDS, +/- antiemetic

Reduce triggers e.g. avoid dietary factors

Prevention

If more than 2 attacks a month, or acute treatment required more than 2x a week

Beta blockers (not for asthmatics)

Tricyclic anti-depressant

Anti-convulsant