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Myocardial infarction, Autorhythmic cells (Conduction cells (L bundle…
Myocardial infarction
Autorhythmic cells
Conduction cells
Purkinje fibers
L bundle branch
Pacemaker cells
SA node
AV bundle
Autorhythmic cells
Conduction cells
R bundle branch
Purkinje fibers
Conduction cells
Ventricular filling
PR interval
Ventricular repolarization
T wave
ST segment
Atrial diastole
Ventricular systole
Ventricular diasole
Ventricular pressure increases
DM increases MI risk
MI damages L ventricle papillary muscles & mitral valve becomes dysfunctional
Upstream effects
60 year old female; History DM 2; MI; Mitral valve not closing; BP over 95/55; Pulse 125; Labored breathing; Gurgling in stethoscope
Background
Blood pathway
Body (systemic circuit)
Superior & inferior vena cava
Right atrium
Tricuspid valve
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Conduction pathway
Cardiac cycle
Ventricular diastole
Artial diastole
Atrial systole
ECG
P wave
Blood pressure
Blood pressure decrease
Stroke volume decreases
Vasoconstriction
Peripheral resistance increases
Afterload increases
Preload decreases
Mean arterial pressure decreases
Cardiac output decreases
Capillary exchange decreases
Blood pressure increase
Mean aterial pressure increases
Stroke volume increases
Vasodilatoin
Afterload decreases
Peripheral resistance decrases
Cardiac output increases
Preload increases
Capillary exchange increases
Downstream effects
Cardiac output decreases
Autonomic nervous system signals the heart to work faster
BP increases
Heart attempts to work harder to recover cardiac output
Damage to the papillary muscles allows back flow through the mitral valve (valve cannot close) reducing the efficiency of the heart
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MAP increases
Pulse increases
Vasoconstriction
Venus return decreases
Stroke volume decreases
Preload decreases
Closing "dub" (S2)
Semilunar valves
Aortic valve
Aorta
Closing "lub" (S1)
AV valves
Mitral valve
Interrnodal pathway
AV node
Ventricular depolarization
QRS complex
Atrial depolarization
Atrial pressure increases
Atrial systole begins
Atrial ejection
Atrial systole ends
AV valve closes
Ventricular systole begins
Isovolumetric contraction
Aortic valve opens
Ventricular ejection
Aortic valve closes
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Atrial pressure decreased
Ventricular pressure decreases
Hypertension
Hypotension
Barorecetpors of the antonomic nervous system
Homeostatic response
Papillary muscles prevent back flow
Source: Fundamentals of Anatomy & Physiology, 11th Edition (class text)