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INNATE IMMUNITY (PAMPS (LPS (CD14 (MO (TLR (CYTOKINES PRODUCED)))),…
INNATE IMMUNITY
PAMPS
LPS
CD14
MO
TLR
CYTOKINES PRODUCED
PEPTYDOGLYCAN WALL
NOD REC
MANNOSE
MANN. BINDING LECITHIN
COMPLEMENT
LIPOTEICHOIC ACID
POSITIVE
dsDNA
VIRUS
UMETHYLATED DNA
BACTERIA
TOLL LIKE RECEPTORS
RECONITION VIA
MAST
DENDRITIC
MO
MACROPHAGES
ACTION
CYTOKINES PRODUCTION
TNF ALFA, IL 1, 6, 8, 12
APC
PEPTIDOGLYCAN
UNMETHYLATED DNA
LPS
CD14
PHAGO
TBC
MODIFIES PHAGOSOME
DIVIDES IN TI
IS PROTECTED FROM AB
CHEDIAK HIGASHI
RESTING
DEBRIS REMOVAL
LYSOZOME
NADPH OXIDAZE
SUPEROXIDES
H202 O2-
MAKES
ENZYMES
NUCLEASAE
LYZOZYME
PROTEASE
RELASE
ABSCESS
ACTIVATION FROM
APC
IFN GAMMA
NK
T CELLS
C5A
SURFACE MOLECULES
CD 14
LPS
B7
CD28
MHC 2
CD4
CD 40
CD40L
C3B
C3B
FC RECE
AB
RELASE
IL
1
ENDOGEN FEVER
INCREASE
SELECTINS
EXPRESSION ON ENDOTHELIUM
8
NO
ATTRACTS
12
TH1
PROMOTES
TNF
ALFA
CACHECTIN
DECREASES USE OF FA
DIC
VASCULAR LEAK
SEPSIS
6
FEVER
CRP
NEUTROPHILS = PMN
BLOOD EXIT
ROLLING
SLOWS DOWN
LIGAND
SIAYL LEWIS 10
E OR P SELECTIN
ON
ENDOTHELIUM
BINDS
NO IS ALWAYS WITH SIAYL
CRAWLING
LPS
CAUSES
INTEGRIN EXPR ON NO
INTEGRIN
ICAM ON ENDOTHELIUM
C5A
MO
SECRETED
TNF
INCREASE IN SELECTINS
ON ENDOTHELIUM
IL 1
TRANSMIGRATION
NO
PECAM 1
BTW ENDOTHELIAL CELLS
MIGRATION
TO INFL
C5A
IL8
CHEMOKINES
ENZYMES
COLLAGENESE
LACTOFERRIN
MYELOPEROXIDASE
RESP BURST
ALP
CML
LOW
INTERAXTIONS
IL8, C5A
CHEMOTAXIS
PHAGO ONLY
IGG
OPSONIN
Fmet
Chemotaxis
BAND
IMMATURE
BACTERIAL INFECTION
GRANULOCYTES
EOSINOPHILS
MPB, ECP, EPO,EDN
ACTIVATION
IL 5
INCREASE IN LVL
IGE
MAST CELLS
TISSUE
IGE
FC
BIND
RELEASE
HISTAMINE
VASODILATION
BASOPHILS
NO
ALL
DESTRUCTIVE ENZYMES
ALL TRIGGERED BY IGE
PARASITES
ALLERGY
NK
MHC 1
ABSENT
KILLS VIRUS INFECTED CELLS
PRODUCE
IFN
ACTIVATES
MO
SURFACE
CD 16
IG G
ADCC
CD 56
NCAM
MARKER OF NK CELLS
COMPLEMENT
C3
ADCC
IG G ON A PATHOGEN
OPSONIN
CD 16 ON NK
NKC KILL
IGE ON PATHOGEN
OPSONIN
EOSINOPHILS
KILL
TH2
ACTIVATION