Meniere's Diseases is characterized during its active phase with the characteristic symptom triad, of episodic vertigo and tinnitus with fluctuations in hearing, followed by a symptom‐free period, ultimately resulting in a more permanent dysfunction of the above symptoms. Any theory attempting to explain the pathophysiology of Meniere's Diseases has to account for processes that result in a reversible dysfunction of both the cochlea and vestibule, with long‐term chronic deficits. Examples of reversible causes include noise, toxins such as salicylates, viral infections and immune‐mediated mechanisms, most of which do not show morphological changes unless they turn permanent.