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DVT Ch 7 - Coggle Diagram
DVT Ch 7
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Virchow's Triad
endothelial injury: change to vascular wall serves as potent thrombotic influence
-directly by surgical trauma
-indirectly by hematoma
-thermal injury by electrocautery or cement polymerization
alteration in blood flow: arterial turbulence or venous stasis contributes to development of thrombi
-stasis occurs on operating table, with post-op immobilization, or impaired ambulation
hypercoagulability: alteration in blood coagulation mechanism that predisposes to thrombi
-transient hypercoagulability may exist as part of normal host response following surgery
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Clinical signs/symptoms
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-calf pain
-swelling
-calf cramping
-warmth
-erythema
-pain along course of involved vein
-engorged veins
-edema
-low-grade fever
-palpable cord along the course of involved vein
When do they develop?
-during surgical procedure
-24-48 hrs post-op
-risk for late post-op DVT recognized to continue for 3 months
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DDx of DVT
-muscle strain
-cellulitis
-superficial thrombophlebitis
-chronic venous insufficiency
-nerve compression syndrome
-lymphedema
-arterial occlusion
-baker cyst
Most often occur in lower extremity, superficial and deep veins
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