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HYPERTENSION, INHIBITION, NO ANGIOTENSIN II, INHIBITION, ORTIZ CORTES…
HYPERTENSION
β-Blockers (BBs)
Blockage of renin release
Vasodilation properties
NO production
Activation of β2 receptors
Blockage of α1 receptors
Blockage of calcium penetration
K+ channels opening
Antioxidant activity
Decrease of heart rate
Decrease of cardiac output
Angiotensin Converting Enzyme Inhibitors (ACEIs)
Conversion of angiotensin I to
angiotensin II
Reduction of vasoconstriction
Decreased blood pressure
Reduction of the concentration of aldosterone
Reduction of the filtration fraction
Stimulation of vasodilator prostaglandins' synthesis
Powerful vasoconstrictor
Increase in blood pressure
Angiotensin II Receptor Blockers (ARBII)
Bindment to AT1 receptors
Complete inhibition of angiotensin II's action
Aldosterone secretion
Reduction of peripheral vascular resistance
Smooth muscle contraction
Vasopressin release
Increase in the sympathetic tone
Adrenal gland's cathecolamine release
Thiazide diuretics
Inhibition of Na+ and Cl- reabsorption
Increased excretion
Decreased vascular volume
Decreased cardiac output
Calcium Channel Blockers (CCBs)
Entrance of calcium to the miocardium and vascular smooth muscle
Decreased vascular muscle tone
Decreased contractility
Decreased heart rate
Decreased cardiac output
Effects on venous vascular tone
Inhibition of calcium transport through the cell membrane
Affection to the arterial vascular tone
INHIBITION
NO ANGIOTENSIN II
INHIBITION
ORTIZ CORTES SILVIA ALITZEL XIADANI
MEDICAL PHARMACOLOGY
UNIVERSIDAD DE GUADALAJARA
CENTRO UNIVERSITARIO DE CIENCIAS DE LA SALUD