Methods for Studying the Brain šŸ‘ØšŸ»ā€āš•ļø

Localist v anti-localist

Localist

(i) The brain is composed of specialised areas. (anatomical evidence)

(ii) Information processing in each of these is local and specific (brain activity studies)

(iii) Discrete areas are responsible for discrete functions (lesion studies)

e.g. Broca

Anti-localist

i) The brain is a collection of networks (anatomical evidence)

(ii) information processing is ā€˜distributed’ (brain activity studies)

(iii) All areas are equally responsible for all functions (lesion studies)

e.g. Flouren's aggregate field theory - small lesions to birds

e.g. Lashley neurons perform all purposes - lesions in rats, size related to impairment not location

Current thinking

localised and distributed processing

evidence for

aims of modern neuroscience

explain functional segregation and functional integration

Insufficiency of lesion studies

Lesions that impair functions

Lesions that don’t impair functions

There are many interpretations to lesion studies so, they cannot tell us about why lesions do/don’t cause deficits.

Essential info processing for the task takes place in that area.

Essential info processing in connected areas is disrupted.

Information processing in that area is not needed for the task.

Information processing is needed for the task in the normal brain, but the lesioned brain compensates to restore function.

Because these alternative explanations are all possibly true, we must study the intact brain.

Brain architecture: connectivity

Diffusion Tensor Imaging

Brain Activity

EEG

MEG

MRI

PET

fMRI

Behavioural neurophysiology

Brain Stimulation

TMS

DBS

Essay q's

Reading

Relationship between neural and cognitive processes

conventional tracer injected into neuron - reveals terminal sites

Info flow is integral part of info processing

can't cross synaptic cleft, can't trace whole circuit

modern tracers

GM, can cross synaptic cleft, reproduce during transmission so signal strength not affected by distance, clefts are labelled so whole circuit is traced

however, only viable in nonhumans

new MRI tech makes it possible to trace fibre pathways

something to do with water diffusion in a fibre bundle

Both human and macaque premotor coretex can be divided into cytoarchitectonically distinct subregions but determining these boundaries in vivo is challenging

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high temporal, low spatial res

ERP's indicate how neural activity changes over time

high temporal, higher spatial because magnetic signal less distorted than EEG

MEG is much more expensive and
physically restrictive than EEG

PET scanners measures changes in blood flow that
are stimulated by changes in neural activity.

ok spatial, poor temporal, can image deep structures as well as cortex, indirect measure

high spatial, low temporal

Important in animal studies for determining exactly where the signal is coming from, at very high temporal spatial res

What is the relationship between the BOLD signal and the
underlying neuronal activity?

Logothetis et al 2001 - BOLD signal

suggests that BOLD activation may actually reflect more the neural activity related to the input and the local processing in any given area, rather than the spiking activity commonly thought of as the output of the area

Both single- and multiunit responses adapt a couple of seconds after stimulus onset, with LFP remaining the only signal correlated with the BOLD response

applied onto cortical areas, the induced current depolarizes nearby located neuron assemblies located beneath the coil and generates neurophysiological and/or behavioral effects depending on their contributing functions

can deliver disruption for a very small amount of time at a very localised area of cortex

can therefore target very specific processesthat occur in particular time points in experiments

Purpose: Stimulation of specific parts of the brain (e.g. specific areas within the basal ganglia) alleviate the symptoms of particular disorders

Method: Electrodes are implanted into specific subcortical areas and connected to implanted stimulators powered by battery

target basal ganglia to treat parkinson's

long-term effects of using highfrequency (130–185 Hz) DBS for Parkinson’s disease are well documented

have shown substantial improvements in symptoms, as measured by motor and daily living scores