Heavy Shadows and Black Night (George Lovell)
Intro
diseases proved to be the most deadly weapon
diseases proved to be the most destructive agent if a fatal complex
some Indian groups fared much better than others in the face of European expansion
the Black legend
It befell an american geographer Carl O Sauer to be among the forms modern scholars to call attention to the impact of the Old World disease on Native American life (1935), a relationship that Las Casas understood well and chose not to emhasize
until the beginning of the 20th century, the Black legend ruled, a view that attributed native loss of life primarily to demoniac acts of cruelty. This was a vision promoted by the Dominican friar Bartolomé de las Casas.
Gibson (1964) overall portrayal is crude and simplistic.
"Who will believe this?" Hispaniola, 1493 - 1518
provoked a lot of heated debate and there is a lot of disagreement
population debates
At the low end, Verlinden (1968) suggests a mere 60000, roughly half the figures put forward by Amiama (1959) and Rosenblat (1976)
at the high end, Cook and Borah (1971) project 8 million.
Sauer, shrewdly uncommitted refers to a figure of 1.100.000 also the number that Las casas mentions (p.428)
Critics of the low number accuse them of defending columbus. Rosenblat (1976) explains the extinction of 10000 instead of 3 million is also not a glorification of colonialism.
1519 what safer says Hispaniola has been reduced to a sorry shell
When diseases broke out on Hispañola
Smallpox
Until recently, the disease factor could not be invoked with confidence because most scholars believed that the first major outbreak of Old World sickness was smallpox, which did not appear in Hispaniola until December 1518 (N. D. Cook and Lovell 1992, 221)
Sauer (1966, 204) called that outbreak "the first epidemic of record."
Henige
Henige (1986, 19) declares that his perusal of sources revealed "no serious or epidemic incidence of infec- tious disease in Hispaniola before late 1518."
there were diseases before
Cook and Borah (1971
there were sick people on the very first voyage and and it is unlikely that the sick would have stayed isolated so that the natives would not have picked up any diseases. They received support from Guerra (1985)
Guerra (1985)
suggests that there was an outbreak of Influenza on Hispañola
unanimity is striking
while the natives suffered infinitely, the invaders were also affected.Of the 1500 men who sailed from Ca'diz on September 25, 1493, scarcely 200 were alive a decade later. Columbus actually himself became ill bur recovered.
influenza transmitted by pigs -> 8 sows had been taken on board -> source of 10 million deaths in 1518 -> then would spread to the continent.
Philippines as a comparison
main difference: native peoples in the Philippines lived alongside domesticated animals, whereas those in the West Indies did not. This meant that humans in the former archipelago had ad- justed biologically to the presence among them of diseases originating in animals, including swine fever.
contact with China from early on also ensured to the old world disease pool
seems like afterwards the Spaniards id not want nothing to do with that area and knew that the riches would be found on the mainland.
Not forever on Earth: Central Mexico 1518 - 1605
its role in the conquest
The Spaniards could never have conquered the city without the support of the diseases. He also admits that the could have never been defeated without the help of the natives. There was a lot of turmoil that broke out because of smallpox, which worked to the invaders advantage.
Here is the text
While the Spaniards were in Tlaxcala, a great plague broke out here in Tenochtitldn. It began to spread during the thirteenth month [September 30-October 19, 1520] and lasted for seventy days, striking everywhere in the city and killing a vast number of our people. Sores erupted on our faces, our breasts, our bellies; we were covered with agonizing sores from head to foot. The illness was so dreadful that no one could walk or move. The sick were so utterly helpless that they could only lie on their beds like corpses, unable to move their limbs or even their heads. They could not lie face down or roll from one side to the other. If they did move their bodies, they screamed with pain. A great many died from this plague, and many others died of hunger. They could not get up to search for food, and everyone else was too sick to care for them, so they starved to death in their beds.6. Look up footnote 6 here and chip it into the essay
scholars agree that the disease described there was smallpox. Disagreement exists over how many perished. (p.430)
the amount of contact population
S. F. Cook and Simpson (1948) first volunteered 11 million for central Mexico, a figure Borah and S. F. Cook (1963) later raised to 25.2 million. Rosenblat (1954) reckoned 4.5 million for all of Mexico, an estimate close to that of Sanders's (1972) 5-6 million inhabitants for the Aztec empire. Sand- ers and archaeologist Barbara Price (1968) favor 12-15 million for all of Mesoamerica. For the Basin of Mexico, Sanders (1976,149) calculates 1-1.2 million
Zambardino
Zambardino concerns himself more with meth- odological procedure than source interpreta- tion, offering a contact figure of 5-10 million for central Mexico, which for him "matches the evidence gathered and presented by Borah and Cook far more accurately than their esti- mate of 25 million
states that all of these numbers contain high margins of error and is in this statement talking about the calculations for the population of Cook and Borah 1971)
Cook and Borah
The Indian population fell by 1620-1625 to a low of approxi- mately 3% of its size at the time the Europeans first landed on the shores of Veracruz. Even if these figures are not true they caused a lot of debate.This school is also called the Berkeley schoo (p.430)
which diseases
first disease is smallpox, we even know the name of the black slave. so there is convergence about the first wave from 1520 onwards, that is not the case for the second wave
difficulties in establishing which diseases we are dealing with
the manner in which a disease is presently thought to behave may not correspond to its manifestation of the past. (2) a second problem is that later outbreaks involved several epidemics.
while the population followed some sudden drops the livestock actually increased. Ecological consequence of the introduction for the animals is still poorly understood
A Long series of encounters, Northwestern Mexico
The North lacked the political and social organization of the South
population
Kroeber contact population is 100000
Reff -> most antive relations were reduced by 30 to over 50%
ome policies of the Spanish contributed to the decline such as mining activity in Durango and southern Chihuahua
Guatemala 1519-32
contact numbers
Zamora -> 315 000. This is in agreement with Solano's tally of 300 000
Denevan, Lovell, Lutz and Swezey: 2 million
Solanos inconsistency
Solano's statistics are more difficult to recon- cile. Even his low contact estimate, when viewed in relation to figures put forward for 1550 and 1575, indicates a 'massive collapse" (1974, 61). For the last quarter of the sixteenth century, however, Solano champions a popula- tion increase that conflicts with abundant con- temporary testimony (Lovell 1992a
Solano (1974, 61) claims that the "Berke- ley School" is driven by "a secret passion," the goal of which is "to blame Spanish actions as the direct cause" of drastic native depopula- tion.
Scattering until they vanished, the central Andes, 1524 - 1635
we know more about the conquest of Mexico because they had adopted a strong written tradition by the time of the conquest. (p.435) For the Incas only few texts exist to illuminate the the principal spanish sources. (p. 435)
The evidence at hand indicates that, as in the case of Guatemala, sickness preceded the physical presence of Spaniards by several years, diffusing ahead of them to weaken mili- tary opposition. An outbreak of what could have been hemorrhagic smallpox, whereby a strain of smallpox infects the blood, causing a rash on the skin similar to that produced by measles, entered the Ecuadorian Andes in 1524 (N. D. Cook 1981, 62; Newson 1992, 88-91).
There it resulted in heavy mortality. Among its victims was the Inca ruler Huayna Capac, who was then in Quito to consolidate Inca power over northern territories recently brought to heel. The epidemic also claimed the life of Huayna Capac's designated heir, igniting a di- sastrous civil war between the brothers. Atahualpa and Huascar, rival contenders for the Inca throne (Dobyns 1963, 496). By the time Pizarro followed up his coastal reconnaissance of the late 1520s with a full-fledged campaign in the 1530s, the chaos that sickness and inter- nal dislocations had brought to Tawantinsuyu facilitated Spanish victory, a fact the invaders themselves openly acknowledged (Wright 1992, 72-75).
origins of smallpox
Atahualpa and Huascar, rival contenders for the Inca throne (Dobyns 1963, 496). By the time Pizarro followed up his coastal reconnaissance of the late 1520s with a full-fledged campaign in the 1530s, the chaos that sickness and inter- nal dislocations had brought to Tawantinsuyu facilitated Spanish victory, a fact the invaders themselves openly acknowledged (Wright 1992, 72-7
There is some disagreement as to the origins of smallpox before it reached the Andes. Most scholars consider passage from Central Amer- ica as the most probable route. Borah (1992, 15), however, suggests a source of contagion among Europeans in the Rfo de la Plata basin, pointing out that the disease in the mountains was reported to have spread from south to north. Newson (1992, 91) resolves the difficulty by arguing that Inca troops stationed in the north near Tumbez may have fallen sick and carried smallpox south to Cuzco, from where it radiated back towards its source of origin. Newson also contends that other diseases could have struck the Incas before Pizarro's arrival; measles and plague are the most likely candidates, Central America the most probable source.
Andean epidemic history has been examined in seminal contributions by Polo (1913), Lastres (1951), and Dobyns (1963), all three of whose work is synthesized by N. D. Cook (1981, 60- 61). After the first outbreak of smallpox, more than twenty different disease episodes took place between 1530-1635, six of them of pan- demic dimension. The cumulative effect of these outbreaks, as in central Mexico, was to decrease native population by the early seven- teenth century to a fraction of its contact size.
Cooks study
While we lack for Tawantinsuyu the plethora of local studies available for Mesoamerica, we are fortunate to have one comprehensive treat- ment of Indian depopulation that compensates in quality for the dearth of regional mono- graphs. N. D. Cook (1981) uses the term "de- mographic collapse" to describe the fate of "Indian Peru" between 1520-1620. In Cook's study, six different methods are either em- ployed or assessed to estimate the size of "Peru's preconquest population." An ecologi- cal or carrying capacity model produces a figure of 6.5 million. Archaeological data, reflecting the poorly developed status of the field compared to Mexico, are considered too inadequate for any kind of calculation beyond those that are site-specific. Even at this level of analysis, however, problems abound: excava- tion at Chan Chan, Cook notes, yields a range of resident occupants from 25,000-200,000. De- population ratio models, believed by Cook to be unreliable because of problems of statistical sampling, generate 6 million (Rowe 1946), 10 million (Wachtel 1977),12 million (Smith 1970), and 37.5 million (Dobyns 1966), all of which are estimates for the central Andes (Ecuador, Peru, Bolivia). Models of political and social struc- ture, an "idealized concept" with "little basis in fact," give a range of 16-32 million (Means 1931, 1932)
census projection models
Census projection models, described as "one of the most promising avenues of ap- proach," deliver a minimum population of 3.9 million and a maximum population of 14.2 mil- lion. Cook's enthusiasm for this procedure, however, does not extend to its manipulation by Shea (1976), whose estimate of 2-2.9 million for the central Andes is dismissed on the grounds of insufficient data and the erroneous supposition "that the rate of decline prior to 1581 paralleled the rate following that date" (N. D. Cook 1981, 95, 108-10).
disease mortality models
Methodologically, perhaps the most novel of Cook's six different strategies is his deploy- ment of disease mortality models, whereby death rates known to have occurred during certain epidemics are applied, with appropriate modification, outbreak by outbreak to the Per- uvian disease chronology. Working from a "cal- culated base" of 671,505 in 1620, the "maximal population" in 1520 is estimated at 8,090,421 and the "minimal population" at 3,243,985. Cook's reasoning is episode-specific: 30-50 percent mortality during the first outbreak of smallpox; 25-30 percent mortality during the first outbreak of measles; and 30-60 percent mortality when smallpox and measles appear together, as they did in the murderous epi- demic of 1585-91, along with mumps, influ- enza, and typhus. The model overlooks many key variables-differential immunity, age- specific mortality, physiological adaptation- but it does offer a reasonable basis for calcula- tion, provided, of course, that disease identification has been established with some degree of confidence (N. D. Cook 1981, 59-74).
conclusion
After dealing, point by point, with the strengths and weaknesses of all six procedures, Cook then steps back from the preponderance of numbers to suggest a specific range (4-15 million) and a specific estimate (9 million) for the population of Peru on the eve of Spanish conquest. These figures are advocated "after careful weighing of the evidence, rather than being purely an act of faith" (1981, 114). The estimate of 9 million people alive in 1520 con- trasts sharply with the estimate of 600,000 alive about a century later. An overall decline of 93 percent "almost completely wiped out" Indi- ans living along the coast. Those who contin- ued to live in the mountains, despite "disease and outright exploitation," in subsequent cen- turies recovered demographically to give the Andes of Peru its unmistakable, enduring na- tive complexion.
general estimation for the population for he Inca empire
Besides his pioneering work at the national level of analysis, which inspired Alchon (1991) toward similar goals in Ecuador, N. D. Cook (1982) has published a population history of the Colca valley that serves as a concrete example of the kind of regional investigation urgently needed throughout the Andes. In the opposite direction, Cook is reported as favoring a con- tact estimate for the entire Inca empire of four- teen million, which means he reckons that some five million people lived under Inca rule in Colombia, Ecuador, Bolivia, Argentina, and Chile (Roberts 1989).
Woman Puma
Woman Puma letter to the king 1200 page -> the Spaniards destroy everything.
The Disease factor in perspective
Population overall: Denevan
posal to arrive at a New World population in 1492 of 57.3 million, which he now adjusts to 53.9 million to incorporate the research findings of the past fifteen years. Denevan's hemispheric estimates are notably higher than the 8.4 million of Kroeber (1939), the 13.4 mil- lion of Rosenblat (1954), and the 15.5 million of Steward (1949). On the other hand, Denevan's reckoning falls far below that of Borah (1976), who mentions upwards of 100 million, and the 90-113 million favored by Dobyns (1966). His estimates most approximate the 37-48.5 mil- lion of Sapper (1924) and the 40-50 million of Spinden (1928). In general, a trend towards acceptance of higher rather than lower contact estimates is apparent, with growing acknowl- edgement that Native American populations a century or so after European intrusion were roughly one-tenth or less their contact size. The disease factor is crucial in any attempt to explain the massive fall in Indian numbers (N. D. Cook and Lovell 1992).
progresss has been made but there is still a lot of resistance
Progress has been made, but much remains to be done. It would be a mistake, for instance, to think that balanced recognition of the role disease played in depopulating the Spanish In- dies is shared by all. This is manifestly not the case, nor is it likely to be. The disease issue is distorted or dodged by all sorts of people for all sorts of reasons, many of them governed more by ideological conviction than by igno- rance or lack of interest. In a study of the plague of 1629 in Muzo, a mining community in Colombia, Friede (1967, 341) declares that "when there were epidemics in Spanish Amer- ica, these were neither general nor of identical consequences throughout the regions af- fected." We may concede Friede the latter point, but he seems unusually blinkered on the former, perhaps influenced too much by the data for the area he happens to know best. For him, however, there exist "numberless docu- ments which definitely attribute the decrease of the Indian population to excessive work, malnutrition, flight, segregation of the sexes, ill-treatment, cruelty, conscription for expedi- tions, enslavement [and the labor draft known as] the mita" (Friede 1967, 339).
Mario Vargas Llosa
Sempat As- sadourian (1985) is of a similar disposition. The mindset is perhaps best exemplified by the Per- uvian writer Mario Vargas Llosa (1990), who manages to address "questions of conquest" in an extended essay in Harper's without once demonstrating awareness that the transfer of disease had a marked influence on how events unfolded.
ultimate conclusion of how disease should be regarded
At the same time as some intellectuals con- veniently ignore or marginalize the disease question, others embrace it far too wholeheart- edly. The trait is particularly evident among some Spanish scholars, whose near-exclusive focus on epidemiology and biological inequal- ity serves to deny that which cannot be denied: barbarous heavy-handedness on the part of the conqueror, from which Indians suffered dread- fully and against which individuals like Las Casas fought and lobbied nobly. Thus we may concur with Zamora (1985, 131) that disease must be considered "the fundamental cause" but express reservation at its being designated the "almost single" cause of native disappear- ance. Similar proclivity to oversimplify the mat- ter may also be found in Guerra (1986, 58), who states with more than a shrug of resignation that "the American Indian was victimized by sickness, not by Spaniards." Historical autopsy should not be performed that surgically, least of all on the countless many who died a con- quered death after October 12, 1492.