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SHOCK (PATHOGENESIS (ENDOTHELIAL DAMAGE, HYPOPERFUSION, INFLAMMATORY…
SHOCK
PATHOGENESIS
ENDOTHELIAL DAMAGE
HYPOPERFUSION
INFLAMMATORY MEDIATORS
CELLULAR SWELLING
INSULT INTRODUCED
EVENTUAL ORGAN DAMAGE
FURTHER SHOCK =TISSUE NECROSIS= DEATH
CLASSIFICATION
CARDIOGENIC
AETIOLOGY IS LOW CO WHICH IS CAUSED BY MYOCARDIAL PUMP FAILURE.
PUMP FAILS DUE TO INFARCTION /OBSTRUCTION
DISTRIBUTIVE
ANAPHYLACTIC
ALLERGIC RXNs= HYPERSENSITIVE RXNs
NEUROGENIC
LOSS VASCULAR TONE DUE TO ANAESTHETIC/ SPINAL CORD INJURY
SEPTIC
SEPTICAEMIA & BACT. ENDOTOXINS
HYPOVOLAEMIC
LOW CO DUE TO [-] BLOOD VOLUME
ENDOCRINE
HYPER/HYPOTHYROIDISM
[+]/[-] INSULIN
HYPOADRENALISM
PITUITARY INFARCT
STAGES
STAGE 2: DECOMPENSATED SHOCK
HYPOTENSION=TISSUE DSYFUNCTION:
CNS=CONFUSION/[-] CONSCIOUSNESS
METABOLIC ACIDOSIS=TACHYPNOEA
LUNG=PULMONARY ODEMA
CAPILLARIES=LEAKY =PERIPHERAL OEDEMA
HEART= BRADYCARDIA
KIDNEY=OLIGURIA
STAGE 3: IRREVERSIBLE SHOCK
TISSUE NECROSIS
LEADS TO MULTI-ORGAN FAILURE
THEN SEVERE ACIDOSIS
THEN HYPOTENSION
THEN BRACHYCARDIA
STAGE 1: COMPENSATED SHOCK
NORMAL BP
CNS FUNCTIONING NORMALLY
TACHYCARDIA & PERIPHERAL VASOCONSTRICTION
EFFECTS OF VASOCONSTRICTION:
BOWEL=NAUSEA
KIDNEY=OLIGURIA
PALE CYNOSIS
DELAYED CAPILLARY REFILL
COLD CLAMMY SKIN
[-] EFFECTIVE CIRCULATING BLOOD VOLUME IMPAIRS TISSUE PERFUSION & LEADS TO CELLULAR HYPOXIA