Chickenpox

Complications

Secondary bacterial infection:
Often group A strep, can be others. May even have further serious complications like nec fasc, TSS syndrome.

Encephalitis. Can occur during the early phase of the illness. Generally good prognosis. Conservative management (i.e. just let it get better by itself). Resolves within a month.

ATAXIA with cerebellar signs (cerebellitis), ~1 week after rash appears

NEW FEVER (fever initially settles and then recurs a few days later = likely 2ndary infection) or PERSISTENT fever after a few days

Purpura fulminans

Cerebellitis, aseptic meningitis, generalised encephalitis

Clinical features

Typical vesicular rash

Illness probably lasts about a week after rash appears, spikes in temperature that wiggle around 40 degrees

Incubation

Rash comes in crops for 3-5 days. Papules >> Vesicles >> Pustules >> Crusts (when fever has come down)

10-23 but median average is 2 weeks. Spread through close contact (15 minutes), respiratory droplets, direct contact with vesicular lesions

Highly infectious during viral shedding - from 2 days before rash appears until it has crusted over

200-500 lesions. Start on the trunk and spread to peripheries

New lesions should not occur after 10 days - suggest defect in cellular immunity

“dew drops on rose petals” appearance

Management

Treatment is symptom-based. No evidence for ORAL acicylovir

Immunocompromised children >> INTRAVENOUS acicylovir

Oral valaciclovir if no organ dissemination. Useful in adults with primary VZV infection (later in life it is worse)

Prophylaxis

Recommended for high risk immunosuppressed/compromised individuals - when they have come into contact with chickenpox infected. Give VZ IV Ig

Children who are immunosuppressed - cancer patients that have received chemotherapy - most viral infections do not pose threat APART from CHICKENPOX and MEASLES

Vasculitis in the skin and soft tissues. Cross reaction of antiviral Abs produced against coagulation factor (protein S) >> dysregulation of clotting, manigestation in the skin.

In the immunocompromised: severe progressive disseminated disease = mortality 20%

In pregnancy

In first 20 w if mother gets primary VZV (not previously immune) Foetus at risk of severe scarring of the skin and possibly ocular and neurological damage and digital dysplasia

Within 5 days of and 2 days after delivery - ,fetus is NOT PROTECTED by maternal antibodies and the viral dose is high. About 25% develop a vesicular rash. The illness has a mortality as high as 30%.

Exposed susceptible mothers can be protected with varicella zoster immune globulin (VZIG) and treated with aciclovir. Infants born in the high-risk period should also receive zoster immune globulin and are often also given aciclovir prophylactically.

Reye syndrome: nausea, vomiting, headache, excitability, delirium, and combativeness with frequent progression to coma

Aspirin use in fever identified as a risk factor for this - stopped using aspirin in children and the rates of Reye syndrome have gone down a lot

Consider varicella vaccination in immunocompromised patients? AVOID LIVE VACCINES. But VARIVAX (for 1yro plus is a live attenuated vaccine)