Burns

Third Degree

Second Degree

First Degree

Emergent

MANIFESTATIONS: dry, waxy white, leathery, or hard skin, insensitivity to pain, possible involvement of muscles, tendons, bones

MANIFESTATIONS: shiny, red fluid filled vesicles, severe pain, mild to moderate edema

Acute:Begins with mobilization of extracellular fluid and subsequent diuresis and ends with wound coverage. :

Rehabiliative Stage of Management(begins when wounds have healed or self care begins).

Pathophysiology

Nursing Assessments

injury to tissue -- duration & temperature --
1st degree: superficial damage to epidermis -- pain and tactile sensation intact
2nd degree: damage to epidermis and dermis --epithelial regeneration skin elements remain
3rd & 4th degree: whole skin element destroyed --destroyed nerve endings -- surgical intervention is required
FLUID SHIFTS: increased permeability --massive fluid shift (water,sodium,plasma proteins) -- decreased colloidal osmotic pressure -- fluid shift into second & third spacing -- insensible fluid loss (30-50 mL/h) -- intravascular volume depletioin -- hypovolemic shock -- hemolysis of RBC -- elevated hematocrit --- major sodium and potassium shift -- restored fluid replacement --- edema ends --interstitial fluid back to vascular space -- diuresis -- coagulation necrosis -- accumulation of neutrophils and monocytes -- wound repair by fibroblasts and collagen fibers -- suseptible to infection

Nursing and Collaborative Management

Complications

Skin and joint contracures, and hypertropic scarring. Contractures will develop due to the shortening of the scar tissue in the flexor tissues of the joint (most at risk: anterior and lateral neck areas, axillae, anticubital fossae, fingers, groin, popliteal fossae, knees and ankles).

Interventions

Complications

Cardiovascular: include dysrhythmias and hypovolemic shock. Circulation to the extremities can be severely impaired. Escharotomy frequently performed.

Other cardiopulmonary problems: patient with preexisting cardiac disease = at high risk for complications. If preexisting respiratory disease = likely to developed respiratory tract infection. Increased risk for VTE.

Respiratory: Upper airway burns and lower airway injury. Watch for signs of impending distress: agitation, anxiety, restlessness or change in patient's breathing.

-Encourage patient and caregiver to participate in care.

Teach patient and caregiver skills for dressing change and wound care.

Arrange home healthcare as needed after discharge

Water-based creams should be used routinely on healed areas to keep the skin moist (decreases itching and flaking). Low dose antihistamines can be used if itching persists.

Urinary: Most common complication is Acute tubular necrosis

Encourage physical and occupational therapy routines. Constant encouragement and reassurance are necessary to maintain the patients morale.

Rehabilitation process is slow and needs to be primary focus for at least the next 6 - 12 months

NURSING ASSESSMENT.
Thermal Buns:
1st Degree: redness, pain , tenderness, minimal edema, blanching with pressure, no vesicles or blisters (although after 24hrs skin may blister and peel)
2nd Degree: Most blebs, blisters, mottled white , pink to cherry red, moderate to severe pain caused by nerve injury, blanching with pressure, red shiny, mild to moderate edema.
3rd Degree: dry, leathery eschar, waxy white, dark brown or charred appearance, strong burn odor, impaired sensation , lack of blanching with pressure
Inhalation burns:
History of being trapped in an enclosed space
Rapid, shallow respirations, increasing hoarseness, coughing, singed nasal or facial hair, smoky breath, difficulty swallowing, restlessness
Electrical Burns:
Leathery, white or charred skin , burn odor, loss of consciousness, impaired touch sensation, dysrhythmias, cardiac arrest, diminished peripheral circulation injured site
Chemical Burns:
Redness, swelling of injured tissue, degeneration of exposed tissue, discoloration of skin, Localized pain, tissue destruction
Respiratory distress, Decreased muscle coordination.

Airway management: Early endotracheal intubation. If intubation not performed administer 100% humidified O2. Place on high fowler's position and encourage deep breathing every hour and reposition patient every 2 hours.

Pathophysiologic changes and manifestations

Skin appears flat and pink. in 4 -6 weeks, the area becomes raised and hyperemic. Mature healing is reached in about 12 months when suppleness has returned and the pink or red color has faded to a slightly lighter color.

Fluid therapy: Crystalloid solutions (Lactated ringer's), colloids (albumin) or combination of two. For the first 24 hours, the recommended is 2 to 4ml lactated ringer's/kg/%TBSA burned

2 characteristics of scarring: discoloration and contour. Discoloration fades with time.

Itching can occur where healing is taking place.

MANIFESTATIONS: erythema, blanching on pressure, pain and mild swelling , no vesicle or blisters

Encourage the use of sunscreen when outside

complications: sodium imbalances, potassium imbalances infection cardiovascular and respiratory complications follow from emergent phase. neurological system changes such as depression, dementia, LOC changes. GI changes, endocrine changes mediated by cortisol.

Possible Causes: Superficial sunburn, Quick heat flash.

Possible Causes:
Flame, Flash, Scald, Contact burns, chemical, Tar, Electric Current

wound care: soap and water or NS to clean the wound silver impregnated dressing. Gauze until after wound has been fully debrided. Blebs, aspiration with tb syringe. Pain management: IV infusions, and prn pain management, anxyolitics. PT and nutritional therapy include antioxidant protocol, multivitamin, high protein, high carb.

Wound care: Debridement. Provide emotional support. Prevent infection. For wound change change dressing every 12 to 24 hours to once every 14 days. Wear protective PPE when changing a wound dressing.

Drug therapy: Analgesics and sedatives to control pain and anxiety. Tetanus immunization (Tetanus toxoid). Antimicrobial agents and covered with light dressing. VTE prophylaxis.

Possible Causes:


Flame, scald, chemicals, tar,
electric current

Other care measures: Eye care for corneal burns or edema with antibiotic ointments. Ears should be kept free from pressure and do not use pillows. For burn hands and arm elevate them on pillows. Keep patient perineum as clean and dry as possible. Monitor fluid and electrolytes. Active and passive exercises to prevent contractures.

Nutritional therapy: early and aggressive nutritional support within several hours of injury. Enteral feedings or parenteral feeding. Feedings slowly at a rate of 20 to 40ml/hr and increase to the goal rate within 24 to 48 hours.